Ophthalmology
Showing 30 of 30 topics
A
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Sudden painful red eye with halos and vision loss; treat immediately with topical and systemic agents to lower IOP; definitive therapy is laser peripheral iridotomy.
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Severe eye pain with halos and nausea; emergent therapy to lower IOP with topical and systemic agents followed by definitive laser iridotomy.
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Severe ocular pain, halos, N/V, and mid-dilated fixed pupil with corneal edema. Begin ED IOP-lowering sequence (topical β-blocker + α-agonist + carbonic anhydrase inhibitor + hyperosmotic agent) and topical pilocarpine once IOP decreased; arrange urgent laser peripheral iridotomy.
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Painful red eye with halos, mid-dilated pupil, corneal edema, and high IOP. Lower pressure with sequential topical and systemic therapy and arrange urgent laser peripheral iridotomy in both eyes when appropriate.
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Severe eye pain, halos, headache, and nausea with mid-dilated fixed pupil and corneal edema. Start immediate pressure-lowering regimen (topical beta-blocker, alpha-agonist, carbonic anhydrase inhibitor; systemic acetazolamide ± hyperosmotic agent), then pilocarpine once IOP lowered; arrange urgent laser iridotomy.
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Pain, photophobia, and ciliary flush with anterior chamber cells require cycloplegic and topical steroid guided by ophthalmology and evaluation for systemic associations.
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Early detection and treatment improve outcomes. Screen at ages 3–5 with visual acuity or instrument‑based screening; treat with refractive correction plus patching or atropine penalization.
B
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Painful red eye with corneal infiltrate and epithelial defect needs urgent ophthalmology, culture for severe or central ulcers, and intensive topical antibiotics.
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Chronic eyelid margin inflammation (anterior staph vs posterior meibomian gland dysfunction). Mainstay is lid hygiene and warm compresses; add topical antibiotics or azithromycin/doxycycline for refractory cases.
C
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Offer cataract surgery when visual function limits daily activities; perform targeted preop assessment including IOL calculations and macular screening; plan for IFIS and anticoagulation management.
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Sudden painless monocular vision loss is an ocular stroke. Activate stroke pathway, assess time last seen normal, control risk factors, and coordinate emergent ophthalmology and stroke services.
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Sudden painless monocular vision loss is an ocular stroke; activate stroke pathway, control risk factors, and urgently involve ophthalmology; consider IOP-lowering measures and hyperbaric oxygen per protocol.
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Sudden painless monocular vision loss is an ocular stroke; activate stroke pathway, consider immediate ocular massage and IOP lowering while arranging urgent ophthalmology and vascular evaluation.
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Sudden, painless monocular vision loss. Treat as an ocular stroke: activate stroke pathway, exclude giant cell arteritis in older patients, consider time-sensitive thrombolysis protocols per center, lower IOP, and start secondary prevention after vascular workup.
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Acute hordeolum is infectious; chalazion is sterile granuloma. Warm compresses are first‑line; incision and curettage or steroid injection for persistent chalazia; antibiotics for external hordeola or preseptal cellulitis.
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Begin copious irrigation immediately with any available isotonic solution, check pH until neutral, remove particulate matter, and arrange urgent ophthalmology.
D
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Anti‑VEGF injections are first‑line for center‑involving DME; choice among aflibercept, ranibizumab, and bevacizumab depends on vision and cost; steroids for pseudophakic or non‑responders.
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Multifactorial disease of tear film and ocular surface. Stage severity; treat with lubricants, anti‑inflammatories (cyclosporine/lifitegrast), lid hygiene/MGD therapy, and punctal occlusion in select cases.
H
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Dendritic epithelial lesions with decreased corneal sensation respond to topical or oral antiviral therapy and avoidance of topical steroid in epithelial disease.
K
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Progressive ectasia causing irregular astigmatism. Diagnose with topography/tomography; halt progression with corneal cross‑linking; use specialty contacts for vision; transplant if advanced.
O
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Visual aura phenomena (scintillations, scotomas) with or without headache. Differentiate from TIA/retinal ischemia; manage triggers and offer migraine prophylaxis when frequent.
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Suspect with peaked pupil, low intraocular pressure, or uveal prolapse. Place rigid shield, avoid pressure and drops, give IV antibiotics and tetanus as indicated, and get urgent ophthalmology.
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Penetrating ocular trauma with peaked pupil, low IOP, or Seidel sign. Avoid any pressure on the eye, place a rigid shield (no patch), give antiemetics and analgesia, start broad IV antibiotics, update tetanus, obtain CT orbit (no tonometry/US), and take urgently to the OR for repair.
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Painful ophthalmoplegia, proptosis, or decreased vision suggests orbital involvement. Start IV antibiotics, obtain urgent imaging, and involve ophthalmology and otolaryngology.
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Painful ophthalmoplegia, proptosis, and vision changes with fever and sinusitis history suggest orbital cellulitis. Start broad IV antibiotics, obtain contrast CT/MRI of orbits/sinuses, and consult ENT/Ophthalmology for abscess drainage when vision threatened or lack of response to antibiotics.
P
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Painful red eye with decreased vision after intraocular surgery needs same day ophthalmology for vitreous tap and intravitreal antibiotics.
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UV‑related fibrovascular growth onto the cornea. Excise when visually significant, symptomatic, or progressive; reduce recurrence with conjunctival autograft ± mitomycin C.
R
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Flashes, floaters, curtain over vision, or new field loss warrant same day ophthalmology to confirm and repair.
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Inherited retinal degeneration causing night blindness and field loss. Provide genetic testing/counseling, manage complications (cataract, CME), and connect to low‑vision services; gene therapy for RPE65 mutations.
T
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Manage with head elevation, eye shield, activity restriction, cycloplegic for comfort, and ophthalmology follow up; check for sickle cell disease in at risk patients.
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