Trauma Surgery
Showing 21 of 21 topics
B
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Recognize primary (barotrauma), secondary (fragmentation), tertiary (displacement), and quaternary effects. Protect providers, manage hemorrhage and airway, and screen for occult pulmonary, ear, and eye injuries.
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Screen with ECG and troponin in blunt chest trauma. If both normal at ~8 hours, significant BCI is unlikely; admit with telemetry if abnormal. Echo for hypotension, arrhythmia, or elevated troponin.
C
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High quality CT can clear the cervical spine in many obtunded adults without neurologic deficit per institutional protocols; maintain collar until clearance is documented.
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Prevent and treat crush syndrome (rhabdomyolysis with hyperkalemia/AKI) with early aggressive fluids, electrolyte management, and careful extrication strategies; monitor for compartment syndrome.
D
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Use staged approach for exsanguinating or physiologically deranged patients with temporary abdominal closure and planned re exploration after resuscitation.
E
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Control hemorrhage with direct pressure or tourniquet, take hard signs to the operating room, and use ABI and CTA for equivocal cases.
F
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Clinical diagnosis after long‑bone or pelvic fractures: triad of hypoxemia, neurologic changes, and petechial rash. Manage with supportive care and early fracture fixation; steroids remain controversial.
G
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Lower physiologic reserve, polypharmacy, and frailty increase mortality. Lower thresholds for activation and imaging; reverse anticoagulation; prioritize early mobilization and delirium prevention.
H
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Urgent assessment of vascularity, nerve function, and tendon integrity. Repair flexor tendons with appropriate core/epitendinous sutures and protect with early controlled motion; repair/neurorrhaphy for nerves with tension‑free technique.
M
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Insert large bore chest tube, resuscitate, and proceed to operative control when output thresholds or instability indicate ongoing bleeding.
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Recognize life threatening hemorrhage and activate massive transfusion with balanced components, early calcium, minimal crystalloid, and timely TXA when indicated.
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Activate MTP early for life-threatening hemorrhage; use balanced component therapy, tranexamic acid within 3 hours, calcium and temperature management, and viscoelastic-guided adjustments.
N
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Differentiate neurogenic from hemorrhagic shock after spinal cord injury; treat with judicious fluids, early vasopressors, and atropine for symptomatic bradycardia.
O
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High mortality injury requiring antibiotics, hemorrhage control, debridement, and fecal or urinary diversion when indicated.
P
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Children differ anatomically and physiologically: larger head, compliant chest, and limited blood reserve. Use weight‑based dosing, minimize radiation, and involve pediatrics early; permissive hypotension is not appropriate.
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Stable patients without peritonitis may be managed selectively with CT, serial exams, and labs; operate for instability, peritonitis, or evisceration.
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Modern approach emphasizes 'no‑zone' evaluation with CTA and airway‑first priorities. Hard signs mandate immediate exploration; otherwise selective management guided by imaging and endoscopy.
T
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Make a clinical diagnosis and decompress immediately using finger or needle thoracostomy, followed by tube thoracostomy.
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Deceleration injury graded I–IV by intimal disruption to rupture. CTA is diagnostic; blood pressure/impulse control with beta‑blockers; TEVAR preferred for grade II+ when anatomy suitable.
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Often occult and left‑sided; maintain high suspicion after blunt or penetrating trauma. CT signs guide diagnosis; laparoscopy/thoracoscopy confirm and repair with non‑absorbable sutures ± mesh for large defects.
U
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Apply pelvic binder at the greater trochanters, exclude other bleeding sources, and proceed to preperitoneal packing, angioembolization, or REBOA based on physiology and resources.
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