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Propofol Infusion Syndrome — Recognition, Prevention, and ICU Management

System: Critical Care • Reviewed: Aug 31, 2025 •

Synopsis:

Critically ill patient on high‑dose or prolonged propofol sedation develops unexplained metabolic acidosis, rhabdomyolysis, acute kidney injury, and cardiac dysfunction—concerning for propofol‑related infusion syndrome (PRIS).

Key Points

  • Suspect PRIS with acidosis + rhabdomyolysis in patients on high‑dose/prolonged propofol.
  • Stop propofol immediately and provide organ‑supportive care.
  • Adhere to dose (<4 mg/kg/h) and duration (<48 h) thresholds when feasible.

Algorithm

  1. Primary survey and stabilization; focused history and exam.
  2. Order high‑yield tests first; escalate imaging as indicated.
  3. Initiate disease‑specific therapy and supportive care.
  4. Reassess clinical response; arrange consultation and disposition.

Clinical Synopsis & Reasoning

Critically ill patient on high‑dose or prolonged propofol sedation develops unexplained metabolic acidosis, rhabdomyolysis, acute kidney injury, and cardiac dysfunction—concerning for propofol‑related infusion syndrome (PRIS). Risk rises with doses ≥4 mg/kg/h or durations >48 h, catecholamines, steroids, and critical illness. Monitor CK, triglycerides, lactate, EKG, renal and hepatic indices. Exclude alternative causes of shock and acidosis.


Treatment Strategy & Disposition

Immediately discontinue propofol; switch to alternative sedatives (e.g., dexmedetomidine, midazolam). Aggressive supportive care: fluids, vasopressors, treat arrhythmias, correct electrolyte derangements; consider renal replacement therapy for severe acidosis/AKI and ECMO for refractory cardiogenic shock. Prevention: adhere to dose/duration limits; avoid combination risk factors where possible.


Epidemiology / Risk Factors

  • Epidemiology varies by setting; see citations for details.

Investigations

TestRole / RationaleTypical FindingsNotes
CK, Lactate, ABGIdentify rhabdomyolysis and acidosis↑CK, metabolic acidosisTrending guides therapy
BMP, LFTsOrgan injury surveillanceAKI, ↑AST/ALT
TriglyceridesMarker and risk signal↑TG
ECG/TelemetryArrhythmia detectionBradyarrhythmias

Pharmacology

MedicationMechanismOnsetRole in TherapyLimitations
Dexmedetomidineα2‑agonistMinutesSedation alternativeBradycardia, hypotension
MidazolamBenzodiazepine GABA‑A agonistMinutesSedation alternativeDelirium, hypotension
Sodium bicarbonateBufferImmediateSevere acidosis (select cases)Paradoxical CNS acidosis, volume load

Prognosis / Complications

  • Prognosis depends on timeliness of diagnosis, comorbid disease, and response to therapy.

Patient Education / Counseling

  • Explain expected course, warning signs requiring urgent care, and follow‑up testing.
  • Review medication use, interactions, and monitoring parameters.

References

  1. Propofol-Related Infusion Syndrome: A Clinical Review (2022) — Link
  2. Propofol infusion syndrome: structured review (2015) — Link
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