Emergency Medicine
Showing 33 of 33 topics
A
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Classify severity by core temperature and mental status; use active external and internal rewarming strategies and anticipate afterdrop and arrhythmias.
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Stage by core temperature; provide gentle handling, airway and circulation support; use passive/active external rewarming and, in severe cases, active internal or extracorporeal rewarming.
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Use symptom triggered or fixed dose benzodiazepines; add phenobarbital or dexmedetomidine as adjuncts in selected severe cases; correct electrolytes and give thiamine.
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Use symptom triggered benzodiazepines guided by CIWA when appropriate; give thiamine before glucose; manage severe or refractory cases with phenobarbital or ICU care.
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Anaphylaxis is life‑threatening; give intramuscular epinephrine immediately in the mid‑anterolateral thigh, repeat as needed, and manage airway/respiration/circulation with adjuncts and observation.
C
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Suspect with headache, dizziness, and exposure history; confirm with COHb level; give high-flow 100% oxygen and consider hyperbaric oxygen for selected indications.
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Use validated decision rules to select imaging; CT is preferred for high risk; clear the collar after adequate imaging and exam when safe.
D
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Use START (adults) and JumpSTART (peds) for mass‑casualty triage to rapidly categorize patients (Immediate/Delayed/Minor/Expectant) based on respiration, perfusion, and mental status.
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Prioritize oxygenation and ventilation, treat hypothermia, and monitor for delayed respiratory compromise; antibiotics are not routinely indicated.
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The BLUE protocol uses lung ultrasound profiles to differentiate causes of acute dyspnea (A‑profile, B‑profile, A’/B’ with DVT, PLAPS). Rapidly narrows diagnosis at bedside.
E
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Ultrasound‑guided nerve blocks provide rapid ED analgesia (e.g., femoral/fascia iliaca for hip fracture, serratus/PECs for rib fractures, forearm blocks for hand injuries). Emphasize safety, dosing, and LAST prevention.
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Assess for arrhythmias, rhabdomyolysis, and occult trauma; obtain ECG and monitor when indicated; manage burns and compartment syndrome risk.
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Anion‑gap ketoacidosis with glucose often <250 mg/dL in patients on SGLT2 inhibitors, during starvation, pregnancy, or perioperatively. Stop SGLT2 agent, initiate fluids, start insulin infusion with concurrent dextrose to clear ketones, correct electrolytes, and address triggers.
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Core temperature ≥40°C with CNS dysfunction. Begin rapid cooling immediately—prefer cold-water immersion for exertional heat stroke or aggressive evaporative cooling with ice packs if immersion unavailable. Avoid antipyretics. Manage rhabdomyolysis, DIC, AKI, and electrolyte derangements; admit to ICU for severe cases.
G
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Risk stratify upper/lower GI bleeding, resuscitate, give PPI for suspected upper GI bleed, reverse anticoagulation as appropriate, and coordinate early endoscopy/consults.
H
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Core temperature ≥40°C with CNS dysfunction; immediate whole-body cold water immersion for exertional cases; evaporative cooling for others; avoid antipyretics.
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Hyperthermia with CNS dysfunction requires immediate whole‑body cold‑water immersion (exertional) or aggressive evaporative cooling; target core 39°C. Manage airway, fluids, rhabdomyolysis, DIC, and organ failure; avoid antipyretics.
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Life threatening hyperthermia with CNS dysfunction; initiate whole body cold water immersion or other rapid cooling and support airway, circulation, and complications.
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Treat ECG changes and K≥6 promptly: stabilize myocardium (calcium), shift K intracellularly (insulin/dextrose, β‑agonist, bicarbonate if acidotic), and remove K (diuresis, binders, dialysis).
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Stabilize myocardium with IV calcium for ECG changes or K ≥6.5; shift K intracellularly (insulin/dextrose, β2-agonist, bicarbonate if acidemic); remove K (diuretics, binders, dialysis).
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BP with acute target‑organ injury (encephalopathy, ACS, APE, aortic dissection, AKI). Reduce MAP by ~20–25% in the first hour (except aortic dissection/ischemic stroke with unique targets) using IV agents (nicardipine, clevidipine, labetalol). Identify/ treat precipitating cause and avoid rapid overcorrection.
P
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Use multimodal analgesia and regional anesthesia to reduce opioid exposure while achieving adequate pain control; provide safe prescribing when opioids are necessary and arrange follow‑up.
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Identify toxidromes (anticholinergic, cholinergic, opioid, sedative‑hypnotic, sympathomimetic, serotonin syndrome). Stabilize ABCs, decontaminate judiciously, give antidotes when indicated, and involve poison control early.
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Preprocedure assessment, fasting considerations, appropriate agent selection, continuous monitoring, and postprocedure recovery criteria reduce complications.
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Ensure medical stability, assess decision‑making capacity and suicide/homicide risk, and follow jurisdictional laws for involuntary holds; document thoroughly and coordinate with psychiatry/social work.
R
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Muscle breakdown with CK elevation causes electrolyte derangements and AKI. Treat with early, high-volume isotonic fluids, correct hyperkalemia, and consider urine alkalinization in select cases; avoid nephrotoxins and monitor urine output/CK trends.
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Muscle injury with marked CK elevation leads to myoglobin-induced AKI and electrolyte disturbances. Initiate early aggressive isotonic fluids, monitor and treat hyperkalemia and hypocalcemia, and consider urine alkalinization selectively; identify and remove precipitating factors.
S
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Identify sepsis early with suspected infection and organ dysfunction. Start broad‑spectrum antibiotics and balanced fluids promptly; obtain cultures and lactate; reassess frequently and source control.
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High‑risk patients with autonomic instability, hallucinations, agitation, or seizures need aggressive benzodiazepine therapy (symptom‑triggered or front‑loaded), early phenobarbital adjunct in refractory cases, thiamine before glucose, electrolyte repletion, and ICU monitoring.
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Obtain ECG and orthostatic vitals on all; identify high-risk features for admission; avoid routine head CT in uncomplicated syncope; tailor workup to suspected cause.
T
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Clinical diagnosis in unstable patients; perform immediate decompression with finger or needle thoracostomy followed by chest tube.
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Clinical diagnosis of life-threatening obstructive shock. Do not wait for imaging: perform immediate needle decompression followed by definitive tube thoracostomy; in monitored settings, prefer finger thoracostomy. Address underlying trauma or barotrauma and reassess ventilation.
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Sudden severe headache peaking within seconds to minutes requires evaluation for subarachnoid hemorrhage with early noncontrast CT and LP or CTA when indicated.
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